June 2015

June 2015

Early Bird Registration Ends July 15 - Register Now

Where: Hyatt Regency San Francisco Airport, Burlingame, CA

When: August 19 - 21, 2015

To Register: http://sens.org/rb2015


Register now for RB2015 to get the best rates. Join SENS Research Foundation, our expert speaker faculty, and a dynamic community of fellow attendees from all across the emerging rejuvenation biotechnology industry. Conference Registration Includes:

  • Full access to all Sessions
  • Full access to Receptions
  • Special Wednesday Night Hal Sparks Performance
  • Continental Breakfast, Lunch and Dinner
  • PM Refreshment Breaks
  • Exhibitor Reception

New Speakers added to 2015 Rejuvenation Biotechnology Conference Roster


SENS Research Foundation would like to extend a warm welcome to the new speakers joining the RB2015 expert faculty this month:

  • Doug Ethell, Head of Molecular Neurobiology, Western University of Health Sciences
  • Sridaran Natesan, Vice President, Strategic Initiatives & Science Relations, Sanofi
  • Stephen Minger, Director, SLM Blue Skies Innovations Ltd.
  • David Brindley, Research Fellow, CASMI Translational Stem Cell Consortium

Visit our website to view the full list of speakers - check back frequently for updates!


RB2015 Sponsorship Packages Available


Support SENS Research Foundation and promote your company at the same time! RB2015 represents an unparalleled opportunity to increase visibility for your business, magazine, or organization. Visit our website for more information and options. 


Why sponsor the 2015 Rejuvenation Biotechnology conference?

  • Position your company as a leader in age-related regenerative medicine
  • Present cutting edge information to your target audience
  • Drive awareness of your organization's current and future products
  • Access world renowned clinicians, researchers, and technologists with a focus on aging and longevity
  • Find collaborators in the age-related disease field

Sign up here

2015 SRF Education Summer Scholars Spotlight: 

Wake Forest Institute for Regenerative Medicine 


The SRF Summer Scholars Program offers undergraduate students the opportunity to conduct biomedical research to combat diseases of aging, such as cancer, Alzheimer’s, and macular degeneration under the guidance of scientific mentors at world-renowned research institutions.


This month we are pleased to introduce our four Summer Scholars stationed at the Wake Forest Institute for Regenerative Medicine: Amanda Paraluppi Bueno, Blake Johnson, Ryan Louer, and Brian Shing. Click each student's photo or name to view more detailed project descriptions on our website.


Amanda Paraluppi Bueno is pursuing a bachelor's degree in Biomedical Science at the Centro Universitario Heminio Ometto, Araras-SP (Brazil). This summer she is working in Dr. Graça Almeida-Porada's lab to help develop novel cell-based therapies that could provide a curative treatment for Inflammatory Bowel Disease (IBD).


Amanda Paraluppi Bueno


Blake Johnson is a senior studying Human Physiology at the University of Iowa. His interest in regenerative medicine was inspired by viewing Dr. Anthony Atala's TED talk on 3-D printed kidney work. This summer, he is working under the direction of Dr. John Jackson to generate thymus organoids capable of producing functional T-cells. 

Blake Johnson


Ryan Louer earned his bachelor's degree in Biochemistry at Purdue University and plans to continue his higher education with the goal of an M.D./Ph.D. He will be working in Dr. Anthony Atala and Dr. James Yoo’s lab under Drs. Myung Jae Jeon and Young Sik Choi this summer studying novel regenerative medicine-based ovarian cell therapies.


Ryan Louer


Brian Shing is a junior at the University of California, Berkeley majoring in Molecular and Cell Biology. Under the mentorship of Drs. In Kap Ko, James Yoo, and Anthony Atala, he will be working on a kidney regeneration project this summer that will explore a novel delivery system allowing for efficient delivery of the secretome secreted from human placental stem cells (hPSCs).


Brian Shing



Question of the Month #11: Are Mitochondrial Mutations Really That Important?


Q: A recent study out of Japan got a lot of coverage in the press, claiming to overthrow much of what was known about the role of mitochondria in aging. It is said to have found that mitochondrial mutations don't really accumulate in aging cells over a lifetime. Instead, it found that age-related mitochondrial dysfunction is driven by epigenetic changes - changes in the scaffolding around DNA that helps regulate which genes are turned on and turned off. In particular, the investigators traced the effect back to the epigenetic downregulation of two genes involved in glycine production in mitochondria, such that providing them with glycine restored much of their normal function. Does this mean mitochondrial mutations really aren't a problem and we can stop working to fix them?
A: The study is interesting, and contributes to a long-standing debate in this field about the frequency of specific mitochondrial DNA mutations with age and tissue type, and whether they contribute to specific diseases. It is clear at this point that michondrial dysfunction occurs with age and that damage in the form of mutations to mitochondria contributes to the diseases and disabilities of aging. We don't believe that this particular study is a challenge to scientists' existing understanding about how changes in mitochondria with age both drive and are driven by cellular and molecular damage, and the diseases and disabilities of aging. To maintain and restore youthful good health in aging people, it remains imperative to repair the cellular and molecular damage of aging directly, including alleviating the effects of large DNA deletions in aging mitochondria.

The "Question of the Month" column is your opportunity to submit your research-related queries to SRF's expert science writer Michael Rae. Please send your questions to foundation@sens.org and they may be featured in a future newsletter.

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